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GWAS Study

The imprinted DLK1-MEG3 gene region on chromosome 14q32.2 alters susceptibility to type 1 diabetes.

Wallace C, Smyth DJ, Maisuria-Armer M et al.

19966805 PubMed ID
GWAS Study Type
29835 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

WC
Wallace C
SD
Smyth DJ
MM
Maisuria-Armer M
WN
Walker NM
TJ
Todd JA
CD
Clayton DG
Chapter II

Abstract

Summary of the research findings

Genome-wide association (GWA) studies to map common disease susceptibility loci have been hugely successful, with over 300 reproducibly associated loci reported to date. However, these studies have not yet provided convincing evidence for any susceptibility locus subject to parent-of-origin effects. Using imputation to extend existing GWA datasets, we have obtained robust evidence at rs941576 for paternally inherited risk of type 1 diabetes (T1D; ratio of allelic effects for paternal versus maternal transmissions = 0.75; 95% confidence interval (CI) = 0.71-0.79). This marker is in the imprinted region of chromosome 14q32.2, which contains the functional candidate gene DLK1. Our meta-analysis also provided support at genome-wide significance for a T1D locus at chromosome 19p13.2. The highest association was at marker rs2304256 (odds ratio (OR) = 0.86; 95%CI = 0.82-0.90) in the TYK2 gene, which has previously been associated with systemic lupus erythematosus and multiple sclerosis.

7,514 European ancestry cases, 9,045 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

29835
Total Participants
GWAS
Study Type
Yes
Replicated
4,840 European ancestry cases, 2,670 European ancestry controls, 5,766 European ancestry familial triads
Replication Participants
European
Ancestry
U.S., U.K.
Recruitment Country
Chapter IV

Analysis

Comprehensive review of health and genetic findings

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