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GWAS Study

Variants within the immunoregulatory CBLB gene are associated with multiple sclerosis.

Sanna S, Pitzalis M, Zoledziewska M et al.

20453840 PubMed ID
GWAS Study Type
5534 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

SS
Sanna S
PM
Pitzalis M
ZM
Zoledziewska M
ZI
Zara I
SC
Sidore C
MR
Murru R
WM
Whalen MB
BF
Busonero F
MA
Maschio A
CG
Costa G
MM
Melis MC
DF
Deidda F
PF
Poddie F
ML
Morelli L
FG
Farina G
LY
Li Y
DM
Dei M
LS
Lai S
MA
Mulas A
CG
Cuccuru G
PE
Porcu E
LL
Liang L
ZP
Zavattari P
ML
Moi L
DE
Deriu E
UM
Urru MF
BM
Bajorek M
SM
Satta MA
CE
Cocco E
FP
Ferrigno P
SS
Sotgiu S
PM
Pugliatti M
TS
Traccis S
AA
Angius A
MM
Melis M
RG
Rosati G
AG
Abecasis GR
UM
Uda M
MM
Marrosu MG
SD
Schlessinger D
CF
Cucca F
Chapter II

Abstract

Summary of the research findings

A genome-wide association scan of approximately 6.6 million genotyped or imputed variants in 882 Sardinian individuals with multiple sclerosis (cases) and 872 controls suggested association of CBLB gene variants with disease, which was confirmed in 1,775 cases and 2,005 controls (rs9657904, overall P = 1.60 x 10(-10), OR = 1.40). CBLB encodes a negative regulator of adaptive immune responses, and mice lacking the ortholog are prone to experimental autoimmune encephalomyelitis, the animal model of multiple sclerosis.

882 Sardinian cases, 872 Sardinian controls

Chapter III

Study Statistics

Key metrics and study information

5534
Total Participants
GWAS
Study Type
Yes
Replicated
1,775 Sardinian cases, 2,005 Sardinian controls
Replication Participants
European
Ancestry
Italy
Recruitment Country
Chapter IV

AI-Generated Summary

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