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GWAS Study

Identification of a sudden cardiac death susceptibility locus at 2q24.2 through genome-wide association in European ancestry individuals.

Arking DE, Junttila MJ, Goyette P et al.

21738491 PubMed ID
GWAS Study Type
35548 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AD
Arking DE
JM
Junttila MJ
GP
Goyette P
HA
Huertas-Vazquez A
EM
Eijgelsheim M
BM
Blom MT
NC
Newton-Cheh C
RK
Reinier K
TC
Teodorescu C
UA
Uy-Evanado A
CN
Carter-Monroe N
KK
Kaikkonen KS
KM
Kortelainen ML
BG
Boucher G
LC
Lagacé C
MA
Moes A
ZX
Zhao X
KF
Kolodgie F
RF
Rivadeneira F
HA
Hofman A
WJ
Witteman JC
UA
Uitterlinden AG
MR
Marsman RF
PR
Pazoki R
BA
Bardai A
KR
Koster RW
DA
Dehghan A
HS
Hwang SJ
BP
Bhatnagar P
PW
Post W
HG
Hilton G
PR
Prineas RJ
LM
Li M
KA
Köttgen A
EG
Ehret G
BE
Boerwinkle E
CJ
Coresh J
KW
Kao WH
PB
Psaty BM
TG
Tomaselli GF
SN
Sotoodehnia N
SD
Siscovick DS
BG
Burke GL
ME
Marbán E
SP
Spooner PM
CL
Cupples LA
JJ
Jui J
GK
Gunson K
KY
Kesäniemi YA
WA
Wilde AA
TJ
Tardif JC
OC
O'Donnell CJ
BC
Bezzina CR
VR
Virmani R
SB
Stricker BH
TH
Tan HL
AC
Albert CM
CA
Chakravarti A
RJ
Rioux JD
HH
Huikuri HV
CS
Chugh SS
Chapter II

Abstract

Summary of the research findings

Sudden cardiac death (SCD) continues to be one of the leading causes of mortality worldwide, with an annual incidence estimated at 250,000-300,000 in the United States and with the vast majority occurring in the setting of coronary disease. We performed a genome-wide association meta-analysis in 1,283 SCD cases and >20,000 control individuals of European ancestry from 5 studies, with follow-up genotyping in up to 3,119 SCD cases and 11,146 controls from 11 European ancestry studies, and identify the BAZ2B locus as associated with SCD (P = 1.8×10(-10)). The risk allele, while ancestral, has a frequency of ~1.4%, suggesting strong negative selection and increases risk for SCD by 1.92-fold per allele (95% CI 1.57-2.34). We also tested the role of 49 SNPs previously implicated in modulating electrocardiographic traits (QRS, QT, and RR intervals). Consistent with epidemiological studies showing increased risk of SCD with prolonged QRS/QT intervals, the interval-prolonging alleles are in aggregate associated with increased risk for SCD (P = 0.006).

1,283 European ancestry cases, ~20,000 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

35548
Total Participants
GWAS
Study Type
Yes
Replicated
3,119 European ancestry cases, 11,146 European ancestry controls
Replication Participants
European
Ancestry
U.S., Netherlands, Finland
Recruitment Country
Chapter IV

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