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GWAS Study

Common variation at 2q22.3 (ZEB2) influences the risk of renal cancer.

Henrion M, Frampton M, Scelo G et al.

23184150 PubMed ID
GWAS Study Type
23306 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HM
Henrion M
FM
Frampton M
SG
Scelo G
PM
Purdue M
YY
Ye Y
BP
Broderick P
RA
Ritchie A
KR
Kaplan R
MA
Meade A
MJ
McKay J
JM
Johansson M
LM
Lathrop M
LJ
Larkin J
RN
Rothman N
WZ
Wang Z
CW
Chow WH
SV
Stevens VL
RD
Ryan Diver W
GS
Gapstur SM
AD
Albanes D
VJ
Virtamo J
WX
Wu X
BP
Brennan P
CS
Chanock S
ET
Eisen T
HR
Houlston RS
Chapter II

Abstract

Summary of the research findings

Genome-wide association studies (GWASs) of renal cell cancer (RCC) have identified four susceptibility loci thus far. To identify an additional RCC common susceptibility locus, we conducted a GWAS and performed a meta-analysis with published GWASs (totalling 2215 cases and 8566 controls of European background) and followed up the most significant association signals [nine single nucleotide polymorphisms (SNPs) in eight genomic regions] in 3739 cases and 8786 controls. A combined analysis identified a novel susceptibility locus mapping to 2q22.3 marked by rs12105918 (P = 1.80 × 10(-8); odds ratio 1.29, 95% CI: 1.18-1.41). The signal localizes to intron 2 of the ZEB2 gene (zinc finger E box-binding homeobox 2). Our findings suggest that genetic variation in ZEB2 influences the risk of RCC. This finding provides further insights into the genetic and biological basis of inherited genetic susceptibility to RCC.

2,215 European ancestry cases, 8,566 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

23306
Total Participants
GWAS
Study Type
Yes
Replicated
3,739 European ancestry cases, 8,786 European ancestry controls
Replication Participants
European
Ancestry
U.S., U.K.
Recruitment Country
Chapter IV

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