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GWAS Study

A cross-platform approach identifies genetic regulators of human metabolism and health.

Lotta LA, Pietzner M, Stewart ID et al.

33414548 PubMed ID
GWAS Study Type
16570 Participants
68 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LL
Lotta LA
PM
Pietzner M
SI
Stewart ID
WL
Wittemans LBL
LC
Li C
BR
Bonelli R
RJ
Raffler J
BE
Biggs EK
OC
Oliver-Williams C
AV
Auyeung VPW
LJ
Luan J
WE
Wheeler E
PE
Paige E
SP
Surendran P
MG
Michelotti GA
SR
Scott RA
BS
Burgess S
ZV
Zuber V
SE
Sanderson E
KA
Koulman A
IF
Imamura F
FN
Forouhi NG
KK
Khaw KT
GJ
Griffin JL
WA
Wood AM
KG
Kastenmüller G
DJ
Danesh J
BA
Butterworth AS
GF
Gribble FM
RF
Reimann F
BM
Bahlo M
FE
Fauman E
WN
Wareham NJ
LC
Langenberg C
Chapter II

Abstract

Summary of the research findings

In cross-platform analyses of 174 metabolites, we identify 499 associations (P < 4.9 × 10-10) characterized by pleiotropy, allelic heterogeneity, large and nonlinear effects and enrichment for nonsynonymous variation. We identify a signal at GLP2R (p.Asp470Asn) shared among higher citrulline levels, body mass index, fasting glucose-dependent insulinotropic peptide and type 2 diabetes, with β-arrestin signaling as the underlying mechanism. Genetically higher serine levels are shown to reduce the likelihood (by 95%) and predict development of macular telangiectasia type 2, a rare degenerative retinal disease. Integration of genomic and small molecule data across platforms enables the discovery of regulators of human metabolism and translation into clinical insights.

16,570 Caucasian ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

16570
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Finland, U.K., Germany
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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