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GWAS Study

Multitrait genetic association analysis identifies 50 new risk loci for gastro-oesophageal reflux, seven new loci for Barrett's oesophagus and provides insights into clinical heterogeneity in reflux diagnosis.

Ong JS, An J, Han X et al.

34187846 PubMed ID
GWAS Study Type
2192831 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

OJ
Ong JS
AJ
An J
HX
Han X
LM
Law MH
NP
Nandakumar P
SJ
Schumacher J
GI
Gockel I
BA
Bohmer A
JJ
Jankowski J
PC
Palles C
OC
Olsen CM
NR
Neale RE
FR
Fitzgerald R
TA
Thrift AP
VT
Vaughan TL
BM
Buas MF
HD
Hinds DA
GP
Gharahkhani P
KB
Kendall BJ
MS
MacGregor S
Chapter II

Abstract

Summary of the research findings

Objective: Gastro-oesophageal reflux disease (GERD) has heterogeneous aetiology primarily attributable to its symptom-based definitions. GERD genome-wide association studies (GWASs) have shown strong genetic overlaps with established risk factors such as obesity and depression. We hypothesised that the shared genetic architecture between GERD and these risk factors can be leveraged to (1) identify new GERD and Barrett's oesophagus (BE) risk loci and (2) explore potentially heterogeneous pathways leading to GERD and oesophageal complications.

129,080 European ancestry cases, 473,524 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

2192831
Total Participants
GWAS
Study Type
Yes
Replicated
462,753 cases, 1,127,474 controls
Replication Participants
European
Ancestry
U.K., Australia, Germany
Recruitment Country
Chapter IV

AI-Generated Summary

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