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GWAS Study

Deciphering osteoarthritis genetics across 826,690 individuals from 9 populations.

Boer CG, Hatzikotoulas K, Southam L et al.

34450027 PubMed ID
GWAS Study Type
890949 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

BC
Boer CG
HK
Hatzikotoulas K
SL
Southam L
SL
Stefánsdóttir L
ZY
Zhang Y
CD
Coutinho de Almeida R
WT
Wu TT
ZJ
Zheng J
HA
Hartley A
TM
Teder-Laving M
SA
Skogholt AH
TC
Terao C
ZE
Zengini E
AG
Alexiadis G
BA
Barysenka A
BG
Bjornsdottir G
GM
Gabrielsen ME
GA
Gilly A
IT
Ingvarsson T
JM
Johnsen MB
JH
Jonsson H
KM
Kloppenburg M
LA
Luetge A
LS
Lund SH
MR
Mägi R
MM
Mangino M
NR
Nelissen RRGHH
SM
Shivakumar M
SJ
Steinberg J
TH
Takuwa H
TL
Thomas LF
TM
Tuerlings M
BG
Babis GC
CJ
Cheung JPY
KJ
Kang JH
KP
Kraft P
LS
Lietman SA
SD
Samartzis D
SP
Slagboom PE
SK
Stefansson K
TU
Thorsteinsdottir U
TJ
Tobias JH
UA
Uitterlinden AG
WB
Winsvold B
ZJ
Zwart JA
DS
Davey Smith G
SP
Sham PC
TG
Thorleifsson G
GT
Gaunt TR
MA
Morris AP
VA
Valdes AM
TA
Tsezou A
CK
Cheah KSE
IS
Ikegawa S
HK
Hveem K
ET
Esko T
WJ
Wilkinson JM
MI
Meulenbelt I
LM
Lee MTM
VM
van Meurs JBJ
SU
Styrkársdóttir U
ZE
Zeggini E
Chapter II

Abstract

Summary of the research findings

Osteoarthritis affects over 300 million people worldwide. Here, we conduct a genome-wide association study meta-analysis across 826,690 individuals (177,517 with osteoarthritis) and identify 100 independently associated risk variants across 11 osteoarthritis phenotypes, 52 of which have not been associated with the disease before. We report thumb and spine osteoarthritis risk variants and identify differences in genetic effects between weight-bearing and non-weight-bearing joints. We identify sex-specific and early age-at-onset osteoarthritis risk loci. We integrate functional genomics data from primary patient tissues (including articular cartilage, subchondral bone, and osteophytic cartilage) and identify high-confidence effector genes. We provide evidence for genetic correlation with phenotypes related to pain, the main disease symptom, and identify likely causal genes linked to neuronal processes. Our results provide insights into key molecular players in disease processes and highlight attractive drug targets to accelerate translation.

up to 400,604 European, Japanese ancestry female cases, up to 490,345 European, Japanese ancestry female controls

Chapter III

Study Statistics

Key metrics and study information

890949
Total Participants
GWAS
Study Type
No
Replicated
East Asian, European
Ancestry
Japan, Greece, Iceland, Norway, U.K., U.S., China
Recruitment Country
Chapter IV

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