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GWAS Study

Genetic variants associated with syncope implicate neural and autonomic processes.

Aegisdottir HM, Thorolfsdottir RB, Sveinbjornsson G et al.

36747475 PubMed ID
GWAS Study Type
1255276 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

AH
Aegisdottir HM
TR
Thorolfsdottir RB
SG
Sveinbjornsson G
SO
Stefansson OA
GB
Gunnarsson B
TV
Tragante V
TG
Thorleifsson G
SL
Stefansdottir L
TT
Thorgeirsson TE
FE
Ferkingstad E
SP
Sulem P
NG
Norddahl G
RG
Rutsdottir G
BK
Banasik K
CA
Christensen AH
MC
Mikkelsen C
PO
Pedersen OB
BS
Brunak S
BM
Bruun MT
EC
Erikstrup C
JR
Jacobsen RL
NK
Nielsen KR
SE
Sørensen E
FM
Frigge ML
HK
Hjorleifsson KE
IE
Ivarsdottir EV
HA
Helgadottir A
GS
Gretarsdottir S
SV
Steinthorsdottir V
OA
Oddsson A
EH
Eggertsson HP
HG
Halldorsson GH
JD
Jones DA
AJ
Anderson JL
KK
Knowlton KU
NL
Nadauld LD
HM
Haraldsson M
TG
Thorgeirsson G
BH
Bundgaard H
AD
Arnar DO
TU
Thorsteinsdottir U
GD
Gudbjartsson DF
OS
Ostrowski SR
HH
Holm H
SK
Stefansson K
Chapter II

Abstract

Summary of the research findings

Aims: Syncope is a common and clinically challenging condition. In this study, the genetics of syncope were investigated to seek knowledge about its pathophysiology and prognostic implications.

56,071 European ancestry cases, 890,790 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

1255276
Total Participants
GWAS
Study Type
Yes
Replicated
22,412 European ancestry cases, 286,003 European ancestry controls
Replication Participants
European
Ancestry
Denmark, U.K., U.S., Finland
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

Independent AI summary of health and genetic findings from the published study

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