Menu
Currency
GWAS Study

Genome-Wide Association Analysis of Protein-Coding Variants in IgA Nephropathy.

Li M, Wang YN, Wang L et al.

37787447 PubMed ID
GWAS Study Type
32017 Participants
74 Views
Scroll to explore
Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LM
Li M
WY
Wang YN
WL
Wang L
MW
Meah WY
SD
Shi DC
HK
Heng KK
WL
Wang L
KC
Khor CC
BJ
Bei JX
CC
Cheng CY
AT
Aung T
LY
Liao YH
CQ
Chen QK
GJ
Gu JR
KY
Kong YZ
LJ
Lee J
CS
Chong SA
SM
Subramaniam M
FJ
Foo JN
CF
Cai FT
JG
Jiang GR
XG
Xu G
WJ
Wan JX
CM
Chen MH
YP
Yin PR
DX
Dong XQ
FS
Feng SZ
TX
Tang XQ
ZZ
Zhong Z
TE
Tan EK
CN
Chen N
ZH
Zhang H
LZ
Liu ZH
TE
Tai ES
LJ
Liu JJ
YX
Yu XQ
Chapter II

Abstract

Summary of the research findings

Significance statement: Genome-wide association studies have identified nearly 20 IgA nephropathy susceptibility loci. However, most nonsynonymous coding variants, particularly ones that occur rarely or at a low frequency, have not been well investigated. The authors performed a chip-based association study of IgA nephropathy in 8529 patients with the disorder and 23,224 controls. They identified a rare variant in the gene encoding vascular endothelial growth factor A (VEGFA) that was significantly associated with a two-fold increased risk of IgA nephropathy, which was further confirmed by sequencing analysis. They also identified a novel common variant in PKD1L3 that was significantly associated with lower haptoglobin protein levels. This study, which was well-powered to detect low-frequency variants with moderate to large effect sizes, helps expand our understanding of the genetic basis of IgA nephropathy susceptibility.

2,378 Han Chinese ancestry cases, 15,642 Han Chinese ancestry controls

Chapter III

Study Statistics

Key metrics and study information

32017
Total Participants
GWAS
Study Type
Yes
Replicated
6,162 Han Chinese ancestry cases, 7,835 Han Chinese ancestry controls
Replication Participants
East Asian
Ancestry
China
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

Independent AI summary of health and genetic findings from the published study

Important: This summary is AI-generated by DNAGENICS for informational purposes only. It was not created by, affiliated with, or endorsed by the researchers behind the original publication, and is based solely on that published research. It may contain errors or omissions. DNAGENICS disclaims all liability for any inaccuracies or consequences arising from use of this information. Verify all information against the original publication. This is not professional scientific review or medical advice.

AI Summary In Progress

Our AI-generated summary of this publication is being prepared. Please check back soon.