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GWAS Study

Genome-wide analysis to assess if heavy alcohol consumption modifies the association between SNPs and pancreatic cancer risk.

Ni Z, Kundu P, McKean DF et al.

38869494 PubMed ID
GWAS Study Type
10134 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

NZ
Ni Z
KP
Kundu P
MD
McKean DF
WW
Wheeler W
AD
Albanes D
AG
Andreotti G
AS
Antwi SO
AA
Arslan AA
BW
Bamlet WR
BL
Beane-Freeman LE
BS
Berndt SI
BP
Bracci PM
BP
Brennan P
BJ
Buring JE
CS
Chanock SJ
GS
Gallinger S
GJ
Gaziano JM
GG
Giles GG
GE
Giovannucci EL
GM
Goggins MG
GP
Goodman PJ
HC
Haiman CA
HM
Hassan MM
HE
Holly EA
HR
Hung RJ
KV
Katzke V
KC
Kooperberg C
KP
Kraft P
LL
LeMarchand L
LD
Li D
MM
McCullough ML
MR
Milne RL
MS
Moore SC
NR
Neale RE
OA
Oberg AL
PA
Patel AV
PU
Peters U
RK
Rabe KG
RH
Risch HA
SX
Shu XO
SK
Smith-Byrne K
VK
Visvanathan K
WJ
Wactawski-Wende J
WE
White E
WB
Wolpin BM
YH
Yu H
ZA
Zeleniuch-Jacquotte A
ZW
Zheng W
ZJ
Zhong J
AL
Amundadottir LT
SR
Stolzenberg-Solomon RZ
KA
Klein AP
Chapter II

Abstract

Summary of the research findings

Pancreatic cancer is a leading cause of cancer-related death globally. Risk factors for pancreatic cancer include common genetic variants and potentially heavy alcohol consumption. We assessed if genetic variants modify the association between heavy alcohol consumption and pancreatic cancer risk.

4,805 European ancestry cases, 5,329 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

10134
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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