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GWAS Study

A genome-wide association study of imaging-defined atherosclerosis.

Gummesson A, Lundmark P, Chen QS et al.

40164586 PubMed ID
GWAS Study Type
26000 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

GA
Gummesson A
LP
Lundmark P
CQ
Chen QS
BE
Björnson E
DK
Dekkers KF
HU
Hammar U
AM
Adiels M
WY
Wang Y
AT
Andersson T
BG
Bergström G
CC
Carlhäll CJ
ED
Erlinge D
JT
Jernberg T
LF
Landfors F
LL
Lind L
MM
Mannila M
MO
Melander O
PC
Pirazzi C
SJ
Sundström J
ÖC
Östgren CJ
GC
Gunnarsson C
OM
Orho-Melander M
SS
Söderberg S
FT
Fall T
GB
Gigante B
Chapter II

Abstract

Summary of the research findings

Imaging-defined atherosclerosis represents an intermediate phenotype of atherosclerotic cardiovascular disease (ASCVD). Genome-wide association studies (GWAS) on directly measured coronary plaques using coronary computed tomography angiography (CCTA) are scarce. In the so far largest population-based cohort with CCTA data, we performed a GWAS on coronary plaque burden as determined by the segment involvement score (SIS) in 24,811 European individuals. We identified 20 significant independent genetic markers for SIS, three of which were found in loci not implicated in ASCVD before. Further GWAS on coronary artery calcification showed similar results to that of SIS, whereas a GWAS on ultrasound-assessed carotid plaques identified both shared and non-shared loci with SIS. In two-sample Mendelian randomization studies using SIS-associated markers in UK Biobank and CARDIoGRAMplusC4D, one extra coronary segment with atherosclerosis corresponded to 1.8-fold increased odds of myocardial infarction. This GWAS data can aid future studies of causal pathways in ASCVD.

26,000 European ancestry individuals

Chapter III

Study Statistics

Key metrics and study information

26000
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Sweden
Recruitment Country
Chapter IV

AI-Generated Summary

AI-generated by DNAGENICS

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