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GWAS Study

Large-scale GWAS of strabismus identifies risk loci and provides support for a link with maternal smoking.

He W, van der Most PJ, Ong JS et al.

40849414 PubMed ID
GWAS Study Type
975385 Participants
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

HW
He W
VD
van der Most PJ
OJ
Ong JS
HL
Hwang LD
WY
Wu Y
MM
Magnø MS
VJ
Vehof J
KK
Krebs K
ML
Mauring L
ÕK
Õunap K
AE
Abner E
MN
Martin NG
PD
Plotnikov D
JC
Jiang C
MR
Melles RB
GP
Gharahkhani P
SH
Snieder H
PT
Palumaa T
KK
Kaljurand K
GJ
Guggenheim JA
MD
Mackey DA
EE
Engle EC
CH
Choquet H
MS
MacGregor S
Chapter II

Abstract

Summary of the research findings

Strabismus is a common pediatric eye misalignment and has complex genetic and environmental causes. Previous genome-wide association studies (GWAS) encountered difficulties in identifying strabismus risk variants due to heterogeneity and small samples. We performed large meta-analyses of 11 European-ancestry GWAS (7 sources), analysing broad strabismus (20,464 cases, 954,921 controls) and subtypes (esotropia/exotropia). We discovered 4 loci (e.g., NPLOC4-TSPAN10-PDE6G-FAAP100, COL6A1) for strabismus and 5 additional loci (e.g., CHRNA4, MAD1L1) for strabismus subtypes and we successfully replicated the previously reported strabismus variant near NPLOC4-TSPAN10-PDE6G-FAAP100. Using mendelian randomisation, we found genetic evidence supporting maternal smoking as a causal risk factor for strabismus in offspring.

20,464 European ancestry cases, 954,921 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

975385
Total Participants
GWAS
Study Type
No
Replicated
European
Ancestry
Netherlands, U.S., Finland, U.K., Australia, Estonia
Recruitment Country
Chapter IV

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