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GWAS Study

Genetically distinct subsets within ANCA-associated vasculitis.

Lyons PA, Rayner TF, Trivedi S et al.

22808956 PubMed ID
GWAS Study Type
9293 Participants
125 Views
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Chapter I

Publication Details

Comprehensive information about this research publication

Authors

LP
Lyons PA
RT
Rayner TF
TS
Trivedi S
HJ
Holle JU
WR
Watts RA
JD
Jayne DR
BB
Baslund B
BP
Brenchley P
BA
Bruchfeld A
CA
Chaudhry AN
CT
Cohen Tervaert JW
DP
Deloukas P
FC
Feighery C
GW
Gross WL
GL
Guillevin L
GI
Gunnarsson I
HL
Harper L
HZ
Hrušková Z
LM
Little MA
MD
Martorana D
NT
Neumann T
OS
Ohlsson S
PS
Padmanabhan S
PC
Pusey CD
SA
Salama AD
SJ
Sanders JS
SC
Savage CO
SM
Segelmark M
SC
Stegeman CA
TV
Tesař V
VA
Vaglio A
WS
Wieczorek S
WB
Wilde B
ZJ
Zwerina J
RA
Rees AJ
CD
Clayton DG
SK
Smith KG
Chapter II

Abstract

Summary of the research findings

Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a severe condition encompassing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis. Its cause is unknown, and there is debate about whether it is a single disease entity and what role ANCA plays in its pathogenesis. We investigated its genetic basis.

914 European ancestry cases, 5,259 European ancestry controls

Chapter III

Study Statistics

Key metrics and study information

9293
Total Participants
GWAS
Study Type
Yes
Replicated
1,454 European ancestry cases, 1,666 European ancestry controls
Replication Participants
European
Ancestry
Czech Republic, Italy, Germany, U.K.
Recruitment Country
Chapter IV

AI-Generated Summary

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